When a puppy is formed in the womb, the hip joint is supposed to form as a ball at the top of the femur (known as the caput) and a shallow bowl-like socket or indentation at the side of the pelvic bone (known as the acetabulum). Covering both the ball and the socket is a layer of cartilage that creates a smooth surface to cushion and protect the joint during movement. Tendons and ligaments surround the joint, holding the two sides together and keeping the ball from dislocating out of the socket.
In somewhere between 15 and 20% of dogs, the hip joint forms abnormally. It is thought that several gene defects contribute to the problem, and environmental problems such as obesity, overexertion, repetitive motion, and injuries probably worsen the situation. Injuries and over-exertion at a very young age are especially problematic, which is why most dog sports do not allow participants younger than six months of age.
Although hip dysplasia can occur in any dog, it is most common in large purebreds including Great Danes, Saint Bernards, Labrador and Golden Retrievers, and German Shepherds. There is no gender prevalence - both sexes are afflicted equally.
There are two common forms of the disease. In the first, the ball portion of the joint isn't seated tightly in the socket, resulting in the hip joint feeling very loose. The second form of hip dysplasia results when the surface of the ball and / or the socket isn't smooth. The instability of the joint or the uneven surface then causes friction upon movement, resulting in joint inflammation.
As the dog walks, the joint becomes inflamed which breaks down the cartilage covering the ball and socket. Inflammation causes more degradation, and it becomes a vicious cycle of inflammation, degradation, inflammation, degradation... Eventually, all of the cartilage is worn away, causing the ball to grind against the socket, bone-on-bone. Known as osteoarthritis, this bone-on-bone movement without any shock-absorbing cartilage in between is quite painful.
The early symptoms of hip dysplasia are rather mild and are generally due to the looseness of the joint. You might notice your dog has a swaying gait and has a hard time standing up. As the disease progresses, the osteoarthritic degeneration causes pain, leading to more advanced symptoms. You will see your dog's activity level decline including an unwillingness to run or jump. Range of motion in the joint will decrease, as will muscle mass in the thighs.
The symptoms of the disease are made worse by the fact that decreased activity often leads to weight gain, which is about the worst thing for a dog with arthritic hips. Poor nutrition is another contributing factor, with giant breed dogs being especially susceptible if food made especially for giant breeds is not used. These dogs grow so fast, it is important to nutritionally support their joints during puppyhood.
The changes related to dysplasia often don't show on an X-ray until the puppy reaches the age of two years, although your dog might show symptoms of pain well before then.
There is no cure for hip dysplasia, but there are several things you can do to help your dog feel better. Physical therapy such as swimming provides passive joint motion which can decrease stiffness while maintaining muscle mass. Carefully monitoring your dog's diet to control his or her weight is the most important home treatment. You might also ask your veterinarian about anti-inflammatory drugs and a pain management regimen. Glucosamine can help the dog maintain the remaining cartilage and even build new cushioning material in the joint.
Mobility aids such as ramps, steps, weight-bearing harnesses, strollers, and wheelchairs are also available to help your dog get around.
When the disease is severe, there are several surgical options that can provide pain relief. For very young dogs (those less than 10 months old) with no evidence of arthritic degeneration, triple pelvic osteotomy, known as TPO, is the surgery of choice. The veterinary surgeon makes three incisions in your dog and cuts the pelvic bone into three sections. The hip sockets are then rotated into a position that allows the ball at the top of the femur to sit more solidly in the socket. Pins are used to maintain the correct alignment. It takes the dog about 3 months to recover, and the surgery has a 90% success rate.
Less commonly, a veterinary surgeon might perform excision arthroplasty to reshape the head of the femur, allowing it to sit more securely in the hip socket.
Finally, for adult dogs who are experiencing severe osteoarthritis, your vet may suggest a total hip replacement, in which both the ball and the socket are replaced with synthetic material. In some cases, the ball portion of the hip may be removed, leaving just the muscles to act as a joint. This is uncommon, but is sometimes done in smaller dogs.
The best way to prevent hip dysplasia is to remove it from the breeding pool. This means that any dog who has hip dysplasia should not be bred, nor should his or her parents. The parents may never show clinical signs of dysplasia, but because the genetics that produce the disease are so complex, it is important that they not be re-bred, even with different partners, after producing even one puppy with dysplasia. What makes this even more complicated is that a puppy may not show symptoms for at least a year or two, and the parents may have been re-bred several times before the breeder even knows of the problem.
When you're interviewing breeders for your new puppy, be sure to ask to see certification that both of the parents have been cleared by a program designed to reduce the incidence of canine hip dysplasia. The most common certifications come from the Orthopedic Foundation for Animals (OFA), the PennHIP program at the University of Pennsylvania, or the British Veterinary Association (BVA).
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